Congenital Visceral Vascular Variation

Received: 03-Mar-2022, Manuscript No. ijav-22-4560; Editor assigned: 06-Mar-2022, Pre QC No. ijav-22-4560(PQ); Reviewed: 11-Mar-2022 QC No. ijav-22-4560; Revised: 17-Mar-2022, Manuscript No. ijav-22-4560(R); Published: 25-Mar-2022, DOI: 10.37532/ijav.2022.15(3).189

Citation: Castellanos B. Congenital Visceral Vascular Variation. Int J Anat Var. 2021;15(3):172-172.

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LETTER

The Variations in the visceral vasculature are frequently encountered, but infrequently beget clinical symptoms. We report a 12- time-old girl with portal hypertension caused by natural variations in visceral vessels. The clinical instantiations included gastrointestinal hemorrhage and ascites. The common hepatic roadway and splenic roadway stem participated the same box from the aorta, and the common hepatic roadway was directly connected with the main portal tone to form an arteriovenous fistula. In addition, the left hepatic roadway and the left gastric roadway participated a common box nominated the “hepatic-gastric box” which began from the anterior wall of the aorta, while the right hepatic roadway began from the superior mesenteric roadway and supplied the right liver. The case was treated with interventional embolization and remained in good condition throughout the follow-up and at the time of publication.

Variations of the visceral vessels have important counteraccusations in cases of liver transplantation, laparoscopic surgery, abdominal radiation intervention, and abdominal trauma. In classical visceral vascular deconstruction (applicable in 64 – 91 of the normal population), the celiac box originates from the anterior wall of the aorta at the position of the first lumbar backbone, and its branches are the common hepatic roadway, the left gastric roadway, and the splenic roadway. Still, rare variation of the abdominal box branches occurs at rates of 8 – 12 [1].

In 1904, handed an embryological explanation for these variations. The 4 caddies on the posterior wall of the primitive abdominal aorta in the embryonic stage are, from superior to inferior left gastric roadway, hepatic roadway, splenic roadway, and superior mesenteric roadway. There are also longitudinal anastomotic highways among them. During embryonic development, the 4 stems gradationally rotate to the frontal side, and concomitantly, the anastomotic highways are intruded and separated. However, it’ll lead to variation, If the gyration is abnormally terminated or the disturbance of the anastomotic highways is interrupted or deficient.

Some scholars have classified these variations. Among them, the groups of Michel’s and Adachi are more traditional, and serve as the marks for all posterior benefactions in this field. In 1928, Adachi deconstructed 252 courses and anatomized variations of the abdominal box, secerning them into six main types and 28 subtypes. Michel’s classified the abdominal box into seven types from 200 necropsies. The most current variations are those of the gastro splenic box and hepatosplenic box [2].

The variations of the abdominal box branches reported in the present case weren’t described in either of the Adachi or Michel’s groups. Still, the variation plant by Demirtas in the deconstruction of a womanish cadaver is veritably analogous one hepatogastric box began from the front of the abdominal aorta and divides into an appurtenant left hepatic roadway and left gastric roadway. Likewise, the other hepatosplenic box began1.5 cm below the hepatogastric box, and divided into the common hepatic roadway and splenic roadway. In a 65-year-old male cadaver, a connection of two anatomical differences was discovered: the first variation concerns the formation of the hepatic portal vein trunk, while the second concerns the branches of the celiac trunk. The inferior and superior mesenteric veins form a common trunk in this scenario, which is then joined to the splenic vein to produce the hepatic portal vein. At the same time, the existence of an incomplete (branched) celiac trunk was revealed, the hepatosplenic trunk from which the common hepatic artery and the splenic artery arise; the left gastric artery arises separately at 0.5 cm supero laterally from the origin of the celiac trunk. Familiarity with this anatomical variation provides useful information for abdominal surgery procedures [3, 4].

The celiac artery or aberrant branches such as a substituted right hepatic artery are responsible for the majority of documented SMA abnormalities. 1 SMA deficiency is quite uncommon. After the paired vitelline arteries fuse during the fifth week of pregnancy, the SMA is produced. The absence of the SMA has been linked to a vascular accident, which could include failure of normal foetal blood vessel resorption, compression or twisting of the mesenteric vasculature, or emboli discharge through placental vascular connections. The majority of the data regarding congenital absence of the SMA is linked to foetal intestinal atresia. Only one incidence of congenitally missing SMA identified in an adult has been recorded thus far [5].

Acknowledgement

The author would like to acknowledge his Department of Anatomy from the University of Touro College of Osteopathic Medicine for their support during this work.

Conflicts of Interest

The author has no known conflicts of interested associated with this paper.

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